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Practice Question

Mr. Anderson is a 50-year-old male client who presented to the emergency department (ED) at 0615 with intermittent cramping abdominal pain, abdominal bloating, and nausea and vomiting for the past 3 days. He denies constipation or diarrhea. He completed his second cycle of chemotherapy infusion last week for a newly diagnosed stage III gastric cancer.

Answer Choices:

Rationale:

🩸Contrast media are nephrotoxic, directly injuring renal tubules and reducing perfusion, leading to AKI.

🚰A large‐volume saline bolus increases intravascular volume and can overload cardiac output, predisposing to pleural effusion.

🚫NPO status causes dehydration and decreased renal blood flow, which is a primary driver of prerenal AKI.

💊Chemotherapy agents can be nephrotoxic and also cause capillary leak syndrome, contributing to both AKI and pleural effusion.

🤢Vomiting leads to ongoing fluid losses and electrolyte disturbances that can precipitate AKI.

📌Core Message

Factors that reduce renal perfusion or are nephrotoxic primarily contribute to acute kidney injury, whereas interventions or conditions that increase intravascular volume predispose to pleural effusion.

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This question is from NCLEX PN-Practice Exam-1 which contains 130 questions.

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Question Details
  • Category: NCLEX PN
  • Subcategory: NCLEX-PN Practice Exams
  • Domain: NCLEX-PN (Practice Exams)
  • Answer Choices: 0
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