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🩸Contrast media are nephrotoxic, directly injuring renal tubules and reducing perfusion, leading to AKI.
🚰A large‐volume saline bolus increases intravascular volume and can overload cardiac output, predisposing to pleural effusion.
🚫NPO status causes dehydration and decreased renal blood flow, which is a primary driver of prerenal AKI.
💊Chemotherapy agents can be nephrotoxic and also cause capillary leak syndrome, contributing to both AKI and pleural effusion.
🤢Vomiting leads to ongoing fluid losses and electrolyte disturbances that can precipitate AKI.
📌Core Message
Factors that reduce renal perfusion or are nephrotoxic primarily contribute to acute kidney injury, whereas interventions or conditions that increase intravascular volume predispose to pleural effusion.
This question is from NCLEX PN-Practice Exam-1 which contains 130 questions.
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